As I understand, natural cardiac vessels dynamically dilate with vigorous exercise. However, stent dilation is static. How does the cardiac system deal with the static effect of a stent.
As I understand, natural cardiac vessels dynamically dilate with vigorous exercise. However, stent dilation is static. How does the cardiac system deal with the static effect of a stent.
Surely you're not suggesting that an atherosclerotic, plaque-filled coronary artery that requires a stent is behaving in a "natural", dynamic way with regards to cardiac demand and output.
That vessel is about as pliable as a cast iron pipe.
Surely you're not suggesting that an atherosclerotic, plaque-filled coronary artery that requires a stent is behaving in a "natural", dynamic way with regards to cardiac demand and output.
That vessel is about as pliable as a cast iron pipe.
To build on this, one the main reasons you’d stent a vessel is because it cannot sufficiently vasodilator to meet blood flow demands, resulting in ischemia. Hence the principle behind a vasodilator stress test or FFR. FFR is a useful tool which, in simple terms, is used to measure just how much the diseased portion of a vessel limits blood flow distally.