- Joined
- Jul 24, 2014
- Messages
- 32
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Here's what I don't understand:
Hypertension --> increased afterload --> decreased stroke volume, cardiac output.... --> decreased blood pressure? normalized blood pressure. --> no problem
or maybe this train of thought
Hypertension --> increased afterload --> decreased stroke volume, cardiac output --> compensatory increase in heart contractility by the symp ns --> maintained stroke volume, cardiac output, exacerbates hypertension. positive feedback loop --> malignant hypertension in like 5 minutes
How does the heart know it must contract harder in response to hypertension-induced increased afterload? Wouldn't parasympathetic tone be increased instead, rather than sympathetic?
Please, my head is spinning in circles.
Thank you in advance!
Hypertension --> increased afterload --> decreased stroke volume, cardiac output.... --> decreased blood pressure? normalized blood pressure. --> no problem
or maybe this train of thought
Hypertension --> increased afterload --> decreased stroke volume, cardiac output --> compensatory increase in heart contractility by the symp ns --> maintained stroke volume, cardiac output, exacerbates hypertension. positive feedback loop --> malignant hypertension in like 5 minutes
How does the heart know it must contract harder in response to hypertension-induced increased afterload? Wouldn't parasympathetic tone be increased instead, rather than sympathetic?
Please, my head is spinning in circles.
Thank you in advance!