Yeah, this stuff is always confusing to me as well. One hormone/electrolyte change leads to another, and the key is knowing at what equilibrium the sequence stops.
My method (not necessarily right), is just projecting the primary changes by the defect, trying to ignore any 2nd or 3rd line consquences (if possible). So with low vitD, you have low Ca (no absorption in the gut), and low P (no (re)absorption in the kidney and gut). PTH will rise (both low Ca and low vitD cause this).
Now the fact that PTH will try to bring up Ca (a SECONDARY effect) is true, but it is not going to be able to compensate (let alone overcompensate). Thus Ca will be low. PTH will also cause P wasting, and this just adds up to the low P caused by lack of vitD (so no ambiguity there).
And yes, a high bone turnover usu. implies high alk phosph.
So I agree with acab.