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We haven't covered this directly yet so I have no clue how it works. I just saw it in FC and figured it would be an opportunity for review for you MS2s.
How does a physiologic level of insulin cause both potassium influx (via Na-K ATPase) and sodium retention in kidneys simultaneously?
How does a physiologic level of insulin cause both potassium influx (via Na-K ATPase) and sodium retention in kidneys simultaneously?