http://lawmedconsultant.com/1986/2m-lesson-in-anesthesia-malpractice
is it hubris? "provider x couldnt intubate you but im sure i can".
is it hubris? "provider x couldnt intubate you but im sure i can".
http://lawmedconsultant.com/1986/2m-lesson-in-anesthesia-malpractice
is it hubris? "provider x couldnt intubate you but im sure i can".
Well, he did intubate the patient successfully for the surgery, so apparently it wasn't hubris unless he traumatized the airway and then extubated in a light plane of anesthesia without checking for a leak around the tube.
Glottic edema from traumatic intubation or oversized tube? or perhaps the real obstruction was subglottic stenosis from the prior trach? Still doesn't really make sense that they couldn't save him with a tracheostomy + epi for bronchospasm. Perhaps the tracheostomy was somehow above a subglottic stenosis (I'm speculating wildly) and that's why the trach didn't work?
Maybe an awake fiberoptic intubation would've been less traumatic and wouldn't have led to glottic edema? Maybe not. Certainly the MDA could've pushed for spinal, but what if the patient absolutely refused?
What is a "MDA"?
tough to comment on a case written up by a legal team... like the telephone game with a toddler.
but, to speculate...
a. regional should not be used as a shield to avoid a difficult airway or comorbidities - these factors should be prepared for and controlled from the get-go. you will eventually get burned...
b. doing a RSI in a patient with the history given sounds negligent - but we weren't there (and it was successful).
c. the primary problem looks to me to have been incomplete neuromuscular blockade reversal. 3 minutes after extubation the trouble began; initially, when masked, sats would recover - sounds like a weak fat guy to me. subsequently, bronchospasm with edema and a difficult airway combined to kill the guy.
was there malpractice? yes, in the case of b. did it kill the guy? i dunno, but it doesn't matter at that point.
interesting/unfortunate case. thanks for posting.
I'm impressed with first the decision for RSI (rather than awake intubation) after that history in an obese,asthmatic, difficult airway. However, the preop airway exam could have been unremarkable (thin neck, MPCL1, full range of motion, nl ) and then due to potential for bronchospasm, desat an RSI could have been appropriate w/ rescue devices (LMA, bourgie, glidescope, FOI, scalpel [you pick]) available.
Now if this patient had a convincing STOP BANG score and was an obese (read: OSA) pt without CPAP therapy prior to elective surgery and insisted on GETA -- then he's an ASA4 perioperatively and either gets a polysomnogram and a course of CPAP or he gets regional for this elective procedure. If he still were to insist and institutionally we "do these cases in these patients all the time", he gets a narcotic free GA without nondepolorizing relaxation and a full disclosure of his anesthetic risks (death, death, trach, death, postop mech. ventilation, sore throat, death, etc.)
"Between 4:00 P.M. and 4:30 P.M., the patients ETCO2 levels remained elevated between 43 and 46. "
-this could be below an OSA pt's baseline PaC02.... how much relaxant and narcotic was given? ... and only 10 minutes since reversal of how many twitches
Throughout all of this he was able to be mask ventilated and oxygenated.... then reintubated, then with difficult oxygenation.
It is at this point that I wonder what could have been done differently to improve this patient's oxygenation (confirm tube placement, albuterol, steroids, rac epi, IV epi, Sevoflurane, theophylline, sacrifices to Jobo?, ECMO/Bypass).
Also pt had a funky epiglottis (? of neg pressure pulm edema from upper airway obstruction)
So the plaintiffs' claim that the defendant did not "immediately recognize and appreciate the patients post-operative respiratory crisis, and immediately re-intubate the patient after initial attempts to ventilate him with a bag and mask failed." --Is this really the issue that lead to this patients demise. The tube did not save this patient rather the inability to oxygenate with a tube in the trachea. Not removing the tube could have been the saving grace for this patient until all narcotics, gas, nondepolarizers, airway edema was out of the picture (especially in light of a prev. difficult airway) (read: sorry sir, the tube just can't come out yet).
Also, when this begins to transpire in the OR, I call the attending surgeon into the room immediately and have him on board from the beginning. He or she will appreciate not having to be brought up to speed while prepping the neck.
Again, the issue perhaps starts at the beginning -- with the anesthetic assessment and planning -- 37 yo prev. diff intubation, obese asthmatic having an elective procedure under GETA most likely with nondepolarizing relaxation and narcotics. (read: hubris) Perhaps this is why the 2 mil was handed over.
Dhan wan
Now if this patient had a convincing STOP BANG score and was an obese (read: OSA) pt without CPAP therapy prior to elective surgery and insisted on GETA -- then he's an ASA4 perioperatively and either gets a polysomnogram and a course of CPAP
ASA Practice Guidelines for the Perioperative Management of Patients with Obstructive Sleep Apnea said:[If a patient is identified as an OSA'er at a preop appointment] the anesthesiologist and surgeon should jointly decide whether to (1) manage the patient perioperatively based on clinical criteria alone or (2) obtain sleep studies, conduct a more extensive airway examination, and initiate indicated OSA treatment in advance of surgery. If this evaluation does not occur until the day of surgery, the surgeon and anesthesiologist together may elect for presumptive management based on clinical criteria or a last-minute delay of surgery.
or he gets regional for this elective procedure. If he still were to insist and institutionally we "do these cases in these patients all the time", he gets a narcotic free GA without nondepolorizing relaxation and a full disclosure of his anesthetic risks
what about the REVERSAL agent used in the case. Could it have precipitated bronchospasm?
I guess we need to also know....which reversal meds did he give (glyco and neostim or was it atropine with glyco)? Also did they push the reversal first followd by the anticholiergic agent (neostig bolus then glyco bolus)? i guess we will never know.....
😉. Also, I don't recommend the atropine/glyco combination you refer to...
Why no nondepolarizers? In this particular case incomplete reversal may have been a factor, but that's not a reason to not use these drugs given an appropriate dose, complete reversal, not extubating before he meets criteria.