3. A 25 yo man just returned from work as worker from Africa begins oral chloroquine therapy for malaria caused by Plasmodium vivax. His initial therapeutic response is good, but he develops recurrent parasitemia 2 months later. Which of the following best explains the recurrence ?
a. Chloroquine is ineffective as oral therapy for P. vivax malaria (WRONG)
b. Chloroquine is ineffective on the exoerythrocytic malaria tissue stages
c. Chloroquine is only effective against P. vivax when combined with metronidazole
d. The patient has a second previously occult malaria infection
e. The patient is inefected with a chloroquine-resistant strain of P.vivax
why is A wrong? chloroquine would not target the hyponozoites, correct? is exoerythrocytic just a fancy name for that stage of hypnozoite? wouldn't the inability to target the hypnozoites make chloroquine ineffective for vivax?
1. a 35 yo woman with infertile, receive injection of contrast material into cervix. on hysterosalpingogram, contrast material also seen in peritoneal cavity, which explain this finding
a) rupture of the fallopian tube
b) rupture of the uterine body
c) spillage of contrast, which an artifact
d) spillage of contrast which normal
why do the tubes spill (d)? Are they normally not connected at all to the ovary? they just float there? This is definitely news to me if so :-O
3. A lab tech wipes doiwn the workbench with alcohol after an experiment. This treatment will successfully inactivated viruses with which of the following characteristics?
A- DNA genome
B- enveloped virion
C- helical capsid
D- icosahedral capsid
E- naked virion
F- RNA genome
33-year-old man dx with epilepsy age 10 years. Most recent generalized tonic-clonic was 5 years ago. Medication was adjusted. Current meds include carbamazepine. He's never had any collisions while driving his motor vehicle. Patient's status with respect to driving?
why is he medically qualified to drive? What is the cutoffs/regulations with this? never heard of this before
5. there was a celiac question.. why did they have pale stools? I thought pale stool tends to happen if you aren't excreting bile into the GI tract, sicne that is what gets converted to stercobillin that gives it the color.
39yo man with polycystic kidney disease has 6 mo hisory of intermitent blood in urine. T 37C, pulse 100, resp 24, BP 160/90. physical shows no other abnormality. his serum urea concentration is 100 mg.ml, creatinine 8mg.dl. urinalysis shows blood. arterial gas would be:
pH pCO2 HCO3
a)7.22/28/11
b)7.32/64/32
c)7.38/40/23
d)7.46/19/13
e)7.49/50/37
I understand why people say A since it was renal failure > metabolic acidosis. I think the way I was thinking about this was that i thought also in PKD people get hypertension from increased renin release due to the cysts blocking blood flow (hence why you give them ACE/ARBs).. so i was thinking E because high RAAS would cause lots of H+ to be excreted, creating the metabolic alkalosis. why is this wrong?
A 40 year old man has had orthostatic hypotension and loose stools for 1 year, 26 years of T1DM. What is causing the loose stools?
Exudation
Malabsorptioni
Motility disorder
Osmosis
Secretoin
Why motility? Somebody said previously it was a neuropathy from diabetes, why wouldn't that cause constipation instead?
5. 18 month old girl with 2 days of cough and hoarseness. 102 fever. Harsh, barking cough. Mild erythema of the oropharyngeal and laryngeal mucosa but no exudate. Rapid strep test is negative. Condition improves within 4 days. What was the cause?
Influenza A, Parainfluenza, RSV, S. aureus, C. diptheriae
I said influenza but it was wrong. Was it RSV? How can I tell what is was just based on "harsh barking cough" I didn't think it was parainfluenza since no inspiratory stridor. I was thinking flu since the erythema in the throat which is where influenza invades, right?
