No, they eat whole grains, fruits, and nuts, albeit not in massive quantities.
Indeed. They also advocate modulating fruit (i.e. carb source) intake according to activity levels. Working out a lot? Have at it. Not so much? Then don't eat so many blueberries..... Pretty simple.
I don't see much extremism here. Though, I haven't looked at the actual research, I can say from personal experience that I do better avoiding grains and other sources of carbs.
Like others have said, you need to individualize. If you're 7-9% bodyfat and do just fine with your current regimine, well, it's pretty hard to argue those results.
However, Paleo aside, I wonder if a LARGE percentage of our society would benefit from some carb restriction given our obesity epidemic as well as, and ofcourse related to, our diabetes epidemic.
Not everyone is gonna be a PGG and stay lean/thin almost regardless. Surely we all know the sort, but when you look around and see nurses waddling back and forth while eating their "low fat" (i.e. high carb) cookies or eating "healthy" spaghetti, all the while getting bigger and bigger and more insulin resistant, perhaps this is telling us something?
Why is the medical community falling so deaf on these issues? Again, we're waiting for people to GET diabetes before we (mostly) start suggesting limiting their carb intake.
We're taking fatties to the OR for gastric bypass and THEN telling them that carbs may not be so great for them..... WTF? Doesn't this epitomize reactionary medicine?
Anything which stimulates insulin is probably not good for weight control. And, it's not calories in calories out. We "forget" that certain foods have different effects on our metabolism, particularly on insulin.
http://biocadmin.otago.ac.nz/fmi/xs...=BIOC2web.fp7&-lay=Lectures&-recid=5263&-find=
Glycolysis
the oxidation of glucose to pyruvate via glucose-6-phosphate with the formation of ATP. Pyruvate is further metabolised by conversion to acetyl-CoA and entry into the TCA cycle with the production of more ATP. Insulin stimulates this pathway by increasing cellular glucose uptake and through the induction of some key enzymes.
Glycogenesis
formation of glycogen from glucose,
stimulated by insulin
Glycogenolysis
breakdown of glycogen to glucose-1-phosphate and thence to glucose-6-phosphate; stimulated by glucagon and adrenalin and
inhibited by insulin. Liver contains the enzyme glucose-6-phosphatase which allows glucose to be formed. Muscle tissues do not contain this enzyme and therefore cannot produce glucose from endogenous glycogen stores.
Gluconeogenesis
formation of glucose from amino acids, lactate and triglyceride-derived glycerol. The conversion of amino acids to glucose is stimulated by cortisol and
inhibited by insulin.
Lipogenesis
formation of triglycerides from glucose,
stimulated by insulin.
Lipolysis
liberation of glycerol and fatty acids from triglycerides, and conversion of glycerol to glucose; lipolysis is
suppressed by insulin, and stimulated by insulin deficiency, glucagon and adrenalin.
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Glucose storage .
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.
[FONT=arial, helvetica]After a carbohydrate meal, the favoured metabolic pathways are glycogenesis and lipogenesis, leading to a increase in the storage of energy as glycogen and triglycerides. This is shown in the diagram above. .
[FONT=arial, helvetica]In the fasting state, glycolysis and lipolysis are favoured, making use of the stored fuels to provide energy for the tissues..