- Joined
- Jun 6, 2019
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Hey everyone,
Interesting case coming up I thought I’d solicit some ideas on.
57 yo F with history of primary pHTN, COPD, OSA non-compliant with CPAP (nocturnal nasal cannula 2L instead), CABG in 2001, DES 4 mo. ago to SVG-RCA. LIMA-LAD patent, otherwise just luminal irregularities and lesions <40%. Scheduled for bilateral venous stasis ulcer debridement at the surgicenter.
No angina. Can “just about” lay flat. But minimal exercise tolerance due to SOB.
Sees pHTN specialist who says she’s optimized on sildenafil TID.
RHC shows PAP 70/44 with systemic BP 108/62.
Echo = Severely dilated/dysfunctional RV. Normal LV, moderate TR but no other valvulopathy.
On Plavix for 4mo. old DES.
The severe pHTN is an exclusion for a case like this to happen at the surgicenter (attached to hospital via covered indoor bridge), but the surgeon told me it is happening there.
On paper, “should be quick,” but with this surgeon, never is.
What’s the plan, Stan?
Interesting case coming up I thought I’d solicit some ideas on.
57 yo F with history of primary pHTN, COPD, OSA non-compliant with CPAP (nocturnal nasal cannula 2L instead), CABG in 2001, DES 4 mo. ago to SVG-RCA. LIMA-LAD patent, otherwise just luminal irregularities and lesions <40%. Scheduled for bilateral venous stasis ulcer debridement at the surgicenter.
No angina. Can “just about” lay flat. But minimal exercise tolerance due to SOB.
Sees pHTN specialist who says she’s optimized on sildenafil TID.
RHC shows PAP 70/44 with systemic BP 108/62.
Echo = Severely dilated/dysfunctional RV. Normal LV, moderate TR but no other valvulopathy.
On Plavix for 4mo. old DES.
The severe pHTN is an exclusion for a case like this to happen at the surgicenter (attached to hospital via covered indoor bridge), but the surgeon told me it is happening there.
On paper, “should be quick,” but with this surgeon, never is.
What’s the plan, Stan?