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In one of their explanations they say:
For a fixed anatomical degree of mitral valve incompetence producing mitral regurgitation and at a given left ventricular preload, an increase in contractility would tend to increase LV afterload.
Maybe I forgot all my physiology, but I can't even find one source that explains why an increases in contractility -> increase in afterload 😕
For a fixed anatomical degree of mitral valve incompetence producing mitral regurgitation and at a given left ventricular preload, an increase in contractility would tend to increase LV afterload.
Maybe I forgot all my physiology, but I can't even find one source that explains why an increases in contractility -> increase in afterload 😕