Lets discuss questions of NBDE 1

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1). .Which angle does a P Wave makes on ECG?
a). .45 degree
b). .180 degree
c). .0 degree
d). .-45 degree
e). .-180 degree.

2). .What is endogenous cholesterol? Most endogenous cholesterol is converted to?
a). .Glucose
b). .Cholic acid
c). .Steroid
d). .Oxaloacetete
e). .Ketone bodies

3). .Which of the following statement is correct regarding Glioblastoma multiforme?
a). .the tumor is most common before puberty
b). .it is classified as a type of meningioma
c). .it is most common type of Astrocytoma.
d). .Its prognosis is generally more favourablethan Grade 1 astrocytoma.
e). .It is derived from the epithelial lining of ventricles

4). .Which of the following pathological changes is irreversible?
a). .fatty changes in liver cells
b). .karyolysis in myocardial cells
c). .glycogen deposition in hepatocyte nuclei
d). .hydropic vacuolization of renal tubular epithelial cells.

5). .An example of Synergism is the effect of?
a). .insulin and glucagon on blood glucose
b). .estrogen and progesterone on uterine motility
c). .growth hormone and thyroxine on skeletal growth.
d). .Antidiuretic hormone and aldosterone on potassium excretion.
 
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which one is correct sequence of spread of depolarization in heart -
1.sa node>av node>bundle branch>atria>ventricles
2.sa node > atria> av node> bundle branch> ventricles
 
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R19 said:
which one is correct sequence of spread of depolarization in heart -
1.sa node>av node>bundle branch>atria>ventricles
2.sa node > atria> av node> bundle branch> ventricles[/QUOTE


Choice 2 👍
Bundle branch --> fine network of Purkinge fibres which penetrate the V myocardium &depolarise the ventricular muscle walls
Click to expand...
 
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Q - 10-year female,what primary teeth would be mobile due to succedaneous eruption?
a) F
b) G
c) H -- key answer
d) I
e) J

why not chices d or e???
isn't at 10 years the eruption of 1st and 2nd premolar?? so their predessors would be about to shed???
 
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atherosclerotic patient would likely involve hypertrophy in what??
a) tunica media
b) intima -- key answer
c) adventitia
d)internal elastic mambrane

why intima??
hypertrophy would result from excessive work of heart agains the constriction caused be the plaque under the mucosa,thats what i know
so what makes the intima hypertrophize????😕
 
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NEAREAST organ to the right kidney?
a/ colon
b/ spleen
c/ stomach
d/ pancreas
e/ duodenum

didnt find anything helpful,any help please ??
 
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teethie said:
Congrats pb2007 for ur score.🙂

Guys, I am done with my exam and it was not an easy exam. But again, it was not very hard too. Basics and bascis is very important to be cleared. otherwise no use of reading multiple materials.
My special thanks to wdent, bratdoc, pb2007, annie(perfectionist1), cindrella, svetlana, elmos,anaita and all others who helped me in clearing the topics.

sdn discussion was very helpful.
guys, focus on your concepts.
Click to expand...

Congrats pb2007 👍
Teethie ..good luck with your results ... i'm sure u r going to do extremely well...👍👍

I still haven't received my scores... its going to take a while :xf:
 
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can anyone just refer a link or an image to study the realtions of lingual artery and nerve with the silivary glands ducts and sublingual muscles
this subject just get me sick and i need a good source to study it
please!!!
 
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Playmaker83 said:
NEAREAST organ to the right kidney?
a/ colon
b/ spleen
c/ stomach
d/ pancreas
e/ duodenum

didnt find anything helpful,any help please ??
Click to expand...

Playmaker83 said:
where did u got that info??
i googled it but didnt find something useful yet😕
Click to expand...
just exclude the wrong options first....spleen,stomach,pancreas are all on left side so cant be the ans.among colon and deodenum,part of colon is located retroperitoneal so it comes nearest to kidneys.
 
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what is the basic principle for dna sequencing by sanger procedure?
translation,transcription,replication or reverse transcription. (i did look up sangers method on wikipedia but didnt understand much😕)
 
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R19 said:
what is the basic principle for dna sequencing by sanger procedure?
translation,transcription,replication or reverse transcription. (i did look up sangers method on wikipedia but didnt understand much😕)
Click to expand...

i go with replication,cz i dont think sanger sequencing has anything with gene expression,its just a method for recording dna sequences,plus it utilizes DNA polymerase,so this is all about replication
i will dig more into it and post any updates🙂
 
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Playmaker83 said:
i go with replication,cz i dont think sanger sequencing has anything with gene expression,its just a method for recording dna sequences,plus it utilizes DNA polymerase,so this is all about replication
i will dig more into it and post any updates🙂
Click to expand...
👍acc to ans key translation should b correct option but lots of ans have been marked wrong in key.do let me know if u find explanation somewhere.
 
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which factor does not affect cusp ht. and fossa depth on restorations?
cuve of spee,intercondylar distance,vertical overlap of ant,horizontal overlap of ant,steepness of articular eminence.

If P waves present in ST segment where does ectopic beats originate??
 
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which of the following calcifyies 1st in ant tooth?
a/ cingulum ... what about this option??
b/ cervical ridge
c/ marginal ridge
d/ incisal ridge ... key answer
e/ root apex

what the right answer,and why?🙂
 
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R19 said:
which factor does not affect cusp ht. and fossa depth on restorations?
cuve of spee,intercondylar distance,vertical overlap of ant,horizontal overlap of ant,steepness of articular eminence.

If P waves present in ST segment where does ectopic beats originate??
Click to expand...

1st question all of the about,but if no chice,am gonna go with intercondylar distance as i found a review mentioning that it doesnt have effect clinically but am trying to support this info with some textbooks

2nd Q, atria,as long as its P wave --> atrial depolaization
 
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Playmaker83 said:
atherosclerotic patient would likely involve hypertrophy in what??
a) tunica media
b) intima -- key answer
c) adventitia
d)internal elastic mambrane

why intima??
hypertrophy would result from excessive work of heart agains the constriction caused be the plaque under the mucosa,thats what i know
so what makes the intima hypertrophize????😕
Click to expand...


You are thinking too far ahead. Yes, there would most likely be some ventricular hypertrophy due to the increased peripheral resistance, but "Atherosclerosis" has its primary effect on the walls of the vessels before the secondary effects are seen in the heart. "Intimal Thickening" is a term that is used to describe the affects associated with the condition; as the plaques are deposited in the vessel wall (a somewhat injurious event), there is intimal thickening which is accompanied a lot of times by actual "Medial Atrophy" (there can also be medial hypertrophy). I would refer you to a pathology/ physiology text for a further explanation, as the process is relatively complex, and can other causes as well.

Note that the Ventricular Hypertrophy is from increased work due to a narrowing of the vessels (decreased compliance), and not from work against the actual atherosclerotic plaques themselves.

http://en.wikipedia.org/wiki/Intima-media_thickness
 
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R19 said:
👍acc to ans key translation should b correct option but lots of ans have been marked wrong in key.do let me know if u find explanation somewhere.
Click to expand...

i think its all about replication,nothing with translation ever,its just a mechanism to obtain the sequence of dna,like DNA finger-print
watch this animation,is really a great simplification of the total process

http://www.dnalc.org/resources/3d/29-sanger-sequencing.html

correct me if u got somthing 🙂
 
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FourthMolar said:
You are thinking too far ahead. Yes, there would most likely be some ventricular hypertrophy due to the increased peripheral resistance, but "Atherosclerosis" has its primary effect on the walls of the vessels before the secondary effects are seen in the heart. "Intimal Thickening" is a term that is used to describe the affects associated with the condition; as the plaques are deposited in the vessel wall (a somewhat injurious event), there is intimal thickening which is accompanied a lot of times by actual "Medial Atrophy" (there can also be medial hypertrophy). I would refer you to a pathology/ physiology text for a further explanation, as the process is relatively complex, and can other causes as well.

Note that the Ventricular Hypertrophy is from increased work due to a narrowing of the vessels (decreased compliance), and not from work against the actual atherosclerotic plaques themselves.

http://en.wikipedia.org/wiki/Intima-media_thickness
Click to expand...

i got it now,what i missed is that the intimal injury,i never thought their would be HYPERTROPHY,as plaaque is deposited under the endothelial lining which later on expand into the lumen and can rupture to produce the other consequences

thanx 4 the explanation 👍
 
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Playmaker83 said:
1st question all of the about,but if no chice,am gonna go with intercondylar distance as i found a review mentioning that it doesnt have effect clinically but am trying to support this info with some textbooks

2nd Q, atria,as long as its P wave --> atrial depolaization
Click to expand...
👍

Playmaker83 said:
which of the following calcifyies 1st in ant tooth?
a/ cingulum ... what about this option??
b/ cervical ridge
c/ marginal ridge
d/ incisal ridge ... key answer👍
e/ root apex

what the right answer,and why?🙂
Click to expand...
d is the ans.
 
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i got my result today, it's 96, i'm very happyyyyyyyyyyyyyyyyyyyyyyy😀
i want to thank everybody, teethie, bratdoc, wdent, pb, cindrella, anaita and everyone helps in this thread.
thanks a lot guys.
best wishes for all.:xf:
 
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elmos said:
i got my result today, it's 96, i'm very happyyyyyyyyyyyyyyyyyyyyyyy😀
i want to thank everybody, teethie, bratdoc, wdent, pb, cindrella, anaita and everyone helps in this thread.
thanks a lot guys.
best wishes for all.:xf:
Click to expand...

Congratulations Elmos on the Great Victory
👍
 
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elmos said:
i got my result today, it's 96, i'm very happyyyyyyyyyyyyyyyyyyyyyyy😀
i want to thank everybody, teethie, bratdoc, wdent, pb, cindrella, anaita and everyone helps in this thread.
thanks a lot guys.
best wishes for all.:xf:
Click to expand...

congrats elmos
i went through the old posts of this thread and i noticed ur name among the contributors who were really good,i got a lot of my concepts cleared according to your posts guys,dont remember all the names,but pb2007,teethie,wdent,cindrella,svetlana and perfectionist1,and some more dont really remember their names
u really deserve it,and best of luck in the admission
 
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R19 said:
👍


d is the ans.
Click to expand...

why D??? calcification starts from DEJ rediating outward,so it follows the same way of matrix deposition,this leads to a conclusion that 1st calcified enamel is at dej below a cusp or a cingulum

correct my concept if wrong please !!!
 
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which anatomic factor restrict amount of lateral shift that occur on working condyle? medial wall of articular fossa on working side or superior wall of articular fossa on non working side??

what is function of primary lysosomes in steroid producing cells??
1.digest cellular debris
2.hydrolyse spent cellular organelle
3.remove excess steroids from cell
4.remove steroid precursors from cells
5.cleave ester from cholesterol ester to produce free cholesterol

does B lymphocytes recognize ag ass with HLA-D??

what happens on loss of canine on 1 side in group function occlusion?
what does atropine do? prevent secretion of ach from autonomic fibres or prevent action of ach on target cell??
 
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elmos said:
i got my result today, it's 96, i'm very happyyyyyyyyyyyyyyyyyyyyyyy😀
i want to thank everybody, teethie, bratdoc, wdent, pb, cindrella, anaita and everyone helps in this thread.
thanks a lot guys.
best wishes for all.:xf:
Click to expand...
Congratulations elmos; an absolutely fabulous score!!! ... feels good to see all of you doing so well in your exam
Hw long was the preparation time?? and what was the pattern of studies you followed??
 
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R19 said:
which anatomic factor restrict amount of lateral shift that occur on working condyle? medial wall of articular fossa on working side or superior wall of articular fossa on non working side?? what other options u got else?? look for tempromandibular ligament or any ligament,cz these 2 is not restricting the lateral shift

what is function of primary lysosomes in steroid producing cells??
1.digest cellular debris --main function of lysosomes
2.hydrolyse spent cellular organelle-- also function of lysosome
3.remove excess steroids from cell -- steroid r not packed in vesicles
4.remove steroid precursors from cells --precursors r not carried out
5.cleave ester from cholesterol ester to produce free cholesterol -- ithink that a function of HDL or LDL(nt sure exactly) in supplying the cell membrane with cholesterol
both 1&2 can be right,

does B lymphocytes recognize ag ass with HLA-D??

what happens on loss of canine on 1 side in group function occlusion? loss of mutually protected occlusion --> increase chance of non-working interference & increase working contact pressure (also it may affect the periodontium of anterior teeth cz they gonna have more work to disclude the posterior teeth ) the between brackets is my conclusion😛
what does atropine do? prevent secretion of ach from autonomic fibres or prevent action of ach on target cell??prevent action of ACH on target cells as its a competitive antagonist of muscarinic receptors,so its gonna block the action of both sypm & parasympathetic systems
Click to expand...

plz correct if wrong
 
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R19 said:
first qn- other options are stylohoid ligament,stylomand ligament and capsular ligament.i dont think any of these is right option.
Click to expand...

i got confused in this question too
but by means of exclusion,i found myself choosing capsular ligament.
the 2nd closest ligament to be true is stylomandibular,but its considered ACCESSORAY to TMJ,then stylohyoid has nothing to do with TMJ
About bones,on working side there's no BONY limitations,which excludes the 1st choice, then on non-working side the condyle moves forward,DOWNWARD and medially,so its actually moving away from the 'superior surface of articular surface'

to add some detail,we all know that the MAJOR restrictors of lateral excursion are:
1/ Working side: temporomandibular ligament
2/ Non-Working side: medial surface of articular fossa

any idea or corrections??
 
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Playmaker83 said:
i got confused in this question too
but by means of exclusion,i found myself choosing capsular ligament.
the 2nd closest ligament to be true is stylomandibular,but its considered ACCESSORAY to TMJ,then stylohyoid has nothing to do with TMJ
About bones,on working side there's no BONY limitations,which excludes the 1st choice, then on non-working side the condyle moves forward,DOWNWARD and medially,so its actually moving away from the 'superior surface of articular surface'

to add some detail,we all know that the MAJOR restrictors of lateral excursion are:
1/ Working side: temporomandibular ligament
2/ Non-Working side: medial surface of articular fossa

any idea or corrections??
Click to expand...
ur right about medial wall of articular fossa as major restictor on non working side.mayb somthin is wrong with qn.capsular ligament's functn is to hold disc in position over condyle,it has nothin to do with lateral shift.
 
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R19 said:
which anatomic factor restrict amount of lateral shift that occur on working condyle? medial wall of articular fossa on working side or superior wall of articular fossa on non working side??

what is function of primary lysosomes in steroid producing cells??
1.digest cellular debris
2.hydrolyse spent cellular organelle
3.remove excess steroids from cell
4.remove steroid precursors from cells
5.cleave ester from cholesterol ester to produce free cholesterol

does B lymphocytes recognize ag ass with HLA-D?? as far as i know,its the T-cell that needs antigen recognition,as B-cell acts on antigens directly and doesnt need it to be expressed by other cells
and also HLA-Dp,q,r are all expressed by B-cells,so how come that they express the Ag and they recognize it

am i thinking right here??


what happens on loss of canine on 1 side in group function occlusion?
what does atropine do? prevent secretion of ach from autonomic fibres or prevent action of ach on target cell??
Click to expand...


🙂
 
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R19 said:
ur right about medial wall of articular fossa as major restictor on non working side.mayb somthin is wrong with qn.capsular ligament's functn is to hold disc in position over condyle,it has nothin to do with lateral shift.
Click to expand...

totally right,but if u take into consideration the capsular attachments,its function would be holding the components of a joint together,this piece of info i believe its in DECKS,so as long as its holding the condyle into articular fossa this means that it prevents the lateral excursions from 'over-extending'
in other words,restricts lateral excursion
 
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Anaita said:
Congratulations elmos; an absolutely fabulous score!!! ... feels good to see all of you doing so well in your exam
Hw long was the preparation time?? and what was the pattern of studies you followed??
Click to expand...

the preparation time was 6 months and half, i studied kaplan book but i didn't do anatomy from there, i did anatomy from nbde first aid and decks,
but for revision i concentrate on my note and nbde first aid, and u have to do first chapter of okesson at least, if u do the first 6 is better.
 
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elmos said:
i got my result today, it's 96, i'm very happyyyyyyyyyyyyyyyyyyyyyyy😀
i want to thank everybody, teethie, bratdoc, wdent, pb, cindrella, anaita and everyone helps in this thread.
thanks a lot guys.
best wishes for all.:xf:
Click to expand...
Congratulations elmos!!!! :bow::bow: awewome score!
 
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Playmaker83 said:
actually it was for me too,but i went thru the antigen stuff og immunity to refresh my info and answered ut Q 😀

how many weeks u have 4 the exam?
Click to expand...
around 5-6 weeks.goin through previous qn papers took so much time🙁 i have to start the next round of whole course again....im worried bout how will i finish whole thing(review book wid decks and asda papers)in such short time...any suggestions??
 
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@simian- on it!
another qn- what are the consequences of sectioning trigeminal nerve below level of medulla??
 
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👍Congratulations elmos, this is the highest score I have heard so far from known ones. This is a real victory and all the very best for your admission.👍👍👍👍👍👍👍



elmos said:
i got my result today, it's 96, i'm very happyyyyyyyyyyyyyyyyyyyyyyy😀
i want to thank everybody, teethie, bratdoc, wdent, pb, cindrella, anaita and everyone helps in this thread.
thanks a lot guys.
best wishes for all.:xf:
Click to expand...
 
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R19 said:
@simian- on it!
another qn- what are the consequences of sectioning trigeminal nerve below level of medulla??
Click to expand...

do decks once,then quit all books and start diggind through everything u think needs some light,and look everywhere (books and web)
then the last 3 weeks for Qs (as much as u can)

about the Q,its lower motor neuron and 2nd order neuron so loss of sensation and motor control on the same side
 
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Playmaker83 said:
do decks once,then quit all books and start diggind through everything u think needs some light,and look everywhere (books and web)
then the last 3 weeks for Qs (as much as u can)

about the Q,its lower motor neuron and 2nd order neuron so loss of sensation and motor control on the same side
Click to expand...
But from what i know lower motor neuron start from spinal cord level(from higher centres till spinal cord is UPM and from spinal cord to periphery is LMN)isn't this right?? so section of nerve below medulla should be UPM after decussation??
 
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