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metabolic disturbance in utero[/QUOTE]
indicates metabolic disturbance during prenatal period. (i didn't know you gave the answer already ^___^) Please answer the question below. =)
question: where is nondisjunction occur most rapidly??? (like what phase -- PMAT?) -- is it the metaphase? when it's lining up? or the Anaphase? when it's separating?
actually small cell carcinoma is associated with para neoplastic syndrome.
ACTH - Cushing's syndrome
ADH- SIADH
PTH.
so all 3 r present in small cell carcinoma. while squamous cell carcinoma of lung is associated with PTH.
(jus checked with an oncologist, he says there ll be hypercalcemia in squamous cell carcnoma of lung,which has metastasized)
was scc also an answer choice in the question in the released paper that u mentioned????
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indicates metabolic disturbance during prenatal period. (i didn't know you gave the answer already ^___^) Please answer the question below. =)
question: where is nondisjunction occur most rapidly??? (like what phase -- PMAT?) -- is it the metaphase? when it's lining up? or the Anaphase? when it's separating?[/quote]
anaphase
isnt it vagus thta follows ECA or CCA in carotid sheath???
1) foll found in epidermis except :
-desmosomes
-capillaries
-mitotic cells
-free nerve endings
2) mucosa of floor of mouth: Does it have a submucosa layer??
3) nerves of anterior abdominal wall lie immediately deep to which layer of wall??
- external oblique
-internal oblique
-transversus abdominis
-transversus fascia
4) stratified squamous epithelium present in??
-jejunum
-stomach
-duodenum
-esophagus
5) in a section of epiphyseal plate of femur of 18 old male;;which is the zone in which cartilage lacunae appear swollen and chondrocytes are large??
-proliferation
-bone deposition
-reserve cartilage
-cartilage calcification
-hypertrophy and maturation
plz help with these questions
thanks
-desmosomes
-capillaries
-mitotic cells
-free nerve endings
2) mucosa of floor of mouth: Does it have a submucosa layer??
3) nerves of anterior abdominal wall lie immediately deep to which layer of wall??
- external oblique
-internal oblique
-transversus abdominis
-transversus fascia
4) stratified squamous epithelium present in??
-jejunum
-stomach
-duodenum
-esophagus
5) in a section of epiphyseal plate of femur of 18 old male;;which is the zone in which cartilage lacunae appear swollen and chondrocytes are large??
-proliferation
-bone deposition
-reserve cartilage
-cartilage calcification
-hypertrophy and maturation
plz help with these questions
thanks
thanks a lot ,,but to the second question i meant whether oral mucosa of floor of mouth is covered with non keratinized epithelium with a lamina propria and submucosa?????
also plz help with the foll:::
1) in respiratory alkalosis ;tetany might be due to sudden decrease in which kind of calcium??
-ionized serum
-citrate bound
-serum protein bound
-total serum
2) P waves occuring in ST segment indicates ectopic beats originating in;
-atria
-AV node
- lower portion of bundle of his
-upper portion of bundle of his
3)what best describes GTP binding proteins?
-tightly bound to adenylate cyclase
-membrane proteins
-inactivate protein kinase C
-component of steroi hormone signal transduction
thanks
also plz help with the foll:::
1) in respiratory alkalosis ;tetany might be due to sudden decrease in which kind of calcium??
-ionized serum
-citrate bound
-serum protein bound
-total serum
2) P waves occuring in ST segment indicates ectopic beats originating in;
-atria
-AV node
- lower portion of bundle of his
-upper portion of bundle of his
3)what best describes GTP binding proteins?
-tightly bound to adenylate cyclase
-membrane proteins
-inactivate protein kinase C
-component of steroi hormone signal transduction
thanks
thanks a lot..
1) cushings syndrome is characterized by foll except one;
- increased protein anabolism
-increased androgen levels
-hyperglycemia
-hypertension
-hyperkalemia
2) cortisol stimulates synthesis in liver of
-glucokinase
-phosphofructokinase
-pyruvate carboxylase
-pyruvate kinase
3)during which phase blood flow to left coronary artery greatest
-late systole
-early systole
-late diastole
-early diastole
thanks
1) cushings syndrome is characterized by foll except one;
- increased protein anabolism
-increased androgen levels
-hyperglycemia
-hypertension
-hyperkalemia
2) cortisol stimulates synthesis in liver of
-glucokinase
-phosphofructokinase
-pyruvate carboxylase
-pyruvate kinase
3)during which phase blood flow to left coronary artery greatest
-late systole
-early systole
-late diastole
-early diastole
thanks
plz help with these questions too::::
1) eg. of innate immunity:
-allergic rxn to insect venom
-classical pathway of complement
-alternative pathway
-destruction of virus infected cells by T killer cells
-production of IgG response to insect venom
2) coagulase production by staphylococus aureus is imp in pathogenicity beoz
-helps to establish a typical abscess lesion
-establish cellulitis lesion
-allows organism to adhere tightly to tissue
-non specifically binds to Fc portion of IgG molecules
3)infection with histoplasma capsulatum in healthy individuals is
-opportunistic infection
-rapidly spreading
-self limiting n benign
4)primary function of S IgA
-aggregates microorganisms and prevents colonization
-activates complement in secretory fluids
-promotes phagocytosis by monocytes
-activates mast cells
-activates NK cells
5) axial inclination of palatal root of max 2nd molar
-distal and lingual
-distal and facial
thanks
1) eg. of innate immunity:
-allergic rxn to insect venom
-classical pathway of complement
-alternative pathway
-destruction of virus infected cells by T killer cells
-production of IgG response to insect venom
2) coagulase production by staphylococus aureus is imp in pathogenicity beoz
-helps to establish a typical abscess lesion
-establish cellulitis lesion
-allows organism to adhere tightly to tissue
-non specifically binds to Fc portion of IgG molecules
3)infection with histoplasma capsulatum in healthy individuals is
-opportunistic infection
-rapidly spreading
-self limiting n benign
4)primary function of S IgA
-aggregates microorganisms and prevents colonization
-activates complement in secretory fluids
-promotes phagocytosis by monocytes
-activates mast cells
-activates NK cells
5) axial inclination of palatal root of max 2nd molar
-distal and lingual
-distal and facial
thanks
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K
kianaz
plz answer
1 polymerization of molecules into collagen fibrile occures in the?
ribosome
lysosome
extracelular space
rough E reticulum
smooth ER
2 injury of spinal accessory while in posterior triangle will cause paralysis of the
platysma
trapezius
sternocleidomastoid
inferrior belly of omohyoid
3 in erythropoiesis which of the following increase from the proerythroblast to the mature erythrocyte?
cell size
phagocytic ability
cytoplasmic acidophilia
cytoplasmic basophilia
thanks
1 polymerization of molecules into collagen fibrile occures in the?
ribosome
lysosome
extracelular space
rough E reticulum
smooth ER
2 injury of spinal accessory while in posterior triangle will cause paralysis of the
platysma
trapezius
sternocleidomastoid
inferrior belly of omohyoid
3 in erythropoiesis which of the following increase from the proerythroblast to the mature erythrocyte?
cell size
phagocytic ability
cytoplasmic acidophilia
cytoplasmic basophilia
thanks
Symptoms include rapid weight gain, particularly of the trunk and face with sparing of the limbs (central obesity), a round face often referred to as a "moon face," excess sweating, telangiectasia (dilation of capillaries), thinning of the skin (which causes easy bruising) and other mucous membranes, purple or red striae (the weight gain in Cushing's stretches the skin, which is thin and weakened, causing it to hemorrhage) on the trunk, buttocks, arms, legs or breasts, proximal muscle weakness (hips, shoulders), and hirsutism (facial male-pattern hair growth). A common sign is the growth of fat pads along the collar bone and on the back of the neck(buffalo hump) (known as a lipodystrophy). The excess cortisol may also affect other endocrine systems and cause, for example, insomnia, reduced libido, impotence, amenorrhoea and infertility. Patients frequently suffer various psychological disturbances, ranging from euphoria to psychosis. Depression and anxiety are also common.[2]
Other signs include polyuria (and accompanying polydipsia), persistent hypertension (due to cortisol's enhancement of epinephrine's vasoconstrictive effect) and insulin resistance (especially common in ectopic ACTH production), leading to hyperglycemia (high blood sugars) which can lead to diabetes mellitus. Untreated Cushing's syndrome can lead to heart disease and increased mortality. Cushing's syndrome due to excess ACTH may also result in hyperpigmentation. This is due to Melanocyte-Stimulating Hormone production as a byproduct of ACTH synthesis from Proopiomelanocortin (POMC). Cortisol can also exhibit mineralcorticoid activity in high concentrations, worsening the hypertension and leading to hypokalemia (common in ectopic ACTH secretion). Furthermore, gastrointestinal disturbances, opportunistic infections and impaired wound healing (cortisol is a stress hormone, so it depresses the immune and inflammatory responses). Osteoporosis is also an issue in Cushing's Syndrome since, as mentioned before, cortisol evokes a stress-like response. The body's maintenance of bone (and other tissues) is therefore no longer one of its main priorities, so to speak. It is also important to note that Cushing's may elicit hirsuitism (male-pattern hair growth in a female) and oligomenorrhea (decreased frequency of menstruation) due to elevations in androgens (male sex hormones), normally at low levels in women.
The syndrome in horses leads to weight loss, polyuria and polydipsia and may cause laminitis
Other signs include polyuria (and accompanying polydipsia), persistent hypertension (due to cortisol's enhancement of epinephrine's vasoconstrictive effect) and insulin resistance (especially common in ectopic ACTH production), leading to hyperglycemia (high blood sugars) which can lead to diabetes mellitus. Untreated Cushing's syndrome can lead to heart disease and increased mortality. Cushing's syndrome due to excess ACTH may also result in hyperpigmentation. This is due to Melanocyte-Stimulating Hormone production as a byproduct of ACTH synthesis from Proopiomelanocortin (POMC). Cortisol can also exhibit mineralcorticoid activity in high concentrations, worsening the hypertension and leading to hypokalemia (common in ectopic ACTH secretion). Furthermore, gastrointestinal disturbances, opportunistic infections and impaired wound healing (cortisol is a stress hormone, so it depresses the immune and inflammatory responses). Osteoporosis is also an issue in Cushing's Syndrome since, as mentioned before, cortisol evokes a stress-like response. The body's maintenance of bone (and other tissues) is therefore no longer one of its main priorities, so to speak. It is also important to note that Cushing's may elicit hirsuitism (male-pattern hair growth in a female) and oligomenorrhea (decreased frequency of menstruation) due to elevations in androgens (male sex hormones), normally at low levels in women.
The syndrome in horses leads to weight loss, polyuria and polydipsia and may cause laminitis
plzz be generous to correct me
please correct me if i am wrong
In 25 yrs male presents with tooth ache,fever,diffuse,firm facial swelling and neutrophilic leukocytosis .the facial swelling most likely represents ?
a cellulitis
b granulation tissue
c eosinophilic infilteration
d granulamatous inflammation
e proliferation of neutrophils
a cellulitis
b granulation tissue
c eosinophilic infilteration
d granulamatous inflammation
e proliferation of neutrophils
answr is sodium in endothial cells. eg: sodium uptake in kidney cells is ative process hance needs lot of atp/energy, hence kidney are very sustible to less oxygen.active transport is NA/K transport,Proton pump/CApump.
i would go for E..just because it is written Neutrophilic leckocytosis.. if someone has better explanation ill be happy to know 🙂
answer1:a.does not produce protein anabolism. cortisols produce protien catabolism n breakdown of muscle proteins.
answer2😛rruvate carboxylase.
answer3:early diastole.
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indicates metabolic disturbance during prenatal period. (i didn't know you gave the answer already ^___^) Please answer the question below. =)
question: where is nondisjunction occur most rapidly??? (like what phase -- PMAT?) -- is it the metaphase? when it's lining up? or the Anaphase? when it's separating?[/QUOTE]
nondsyjunction occur during separation of sister chromatid away from the eqitoral plane after exchange og genetic material.
diffuse facial sweeling means it is not localized or is rapildy sdpreading. hence it cannot be grnulationgranulomatous inflammation where the swelling is soft due to accumalation of pus .
facial sweeling with systemic manifestations like fever are highly indicative of cellulitis . MOST COMMON COMPLICATION OF SPREAD OF ORAL INFECTIONS VIA FACIAL SPACES.
Guys i am not sure for this answer if anybody has the answer please enlighten us . in think so that is cellulitis
😕the branches of external carotidwhich are not found in the carotid triangle
1. superficial temporal
2. maxillary
3. posterior superior alveolar artery
pleasde correct me if i am wrong
when an uncoupler is added then it function in such a way that though a proton gradient is MAINED across the mitochondrial membrane but no electron are combined with oxygen.. theULTIMATE STEP IN OXIDATIVE PHOSPHORYLATION wherein ATP sysnthesis is clubed with electron transfer which in presence of O2 form water n co2 final byproducts of respiration. UNCOUPLERS use the neregy of electron transport for generation of heat instead OF GENEARATION OF ATP.Hence no atp/no energy generation but body is heated.Same mechanism is utlized during shivering.or high aspirin doses.
hey!!!
POLYMERISATION OF PROCOLLAGEN MEAN ADDITON OF NEW AMINOACIDS TO THE PREEXISTING CHAIN. (JUST LIKE POLYMERISATION OF ETHELENE OR ANY OTHER STUFF.. PLYMERISATION IS JUST ADDITON).Hence additon n chain elongation takes place precisely in RER. golgi is associated only with pacakage n distributuion.I m pretty sure.
Guys, I also have some questions that I need help with please!
Baroreceptors work to decrease BP by:
Inc Parasymp innerv to Blood vessels
or
Inc PS innerv to the Heart?
What hormone is released in response to Histamine relase?
Would delayed closing of Na channels lead to a lingering + charge after an AP?
(if no, what would cause that?)
What metabolism does Clostridium Perfringens utilize?
If you had cell damage, would you see an increase in conc of K or an increase in conc of Na?
What does St Louis encephalopathy cause?
Are there any other Aids-associated neoplasms besides CNS lymphoma and Kaposi's sarcoma? (I'm sure there are, I just dont know what they are!)
Please help, Thank you in advance!
Baroreceptors work to decrease BP by:
Inc Parasymp innerv to Blood vessels
or
Inc PS innerv to the Heart?
What hormone is released in response to Histamine relase?
Would delayed closing of Na channels lead to a lingering + charge after an AP?
(if no, what would cause that?)
What metabolism does Clostridium Perfringens utilize?
If you had cell damage, would you see an increase in conc of K or an increase in conc of Na?
What does St Louis encephalopathy cause?
Are there any other Aids-associated neoplasms besides CNS lymphoma and Kaposi's sarcoma? (I'm sure there are, I just dont know what they are!)
Please help, Thank you in advance!
good luck on the exam,,when r u taking it and from where did u get the Qs?
What hormone is released in response to Histamine relase?
histamine-->vasoldilation-->low gfr--> renin angiotensin Aldoesteron axis is activated..
histamine-->vasoldilation-->low gfr--> renin angiotensin Aldoesteron axis is activated..
1) general labial convexity seen in ???? is it mand incisors??
2) greater overjet of anterior teeth then::
-shorter the cusps of posterior teeth
-greater intercondylar distance
-lesser intercondylar distance
-more acute benett angle
-more obtuse benett angle
3)internal carotid arteries become occluded then blood reach arterial circle via:
-vertebral artery???
4) role of calcium in activation of cardiac muscle
-couple myosin n actin to form actinomyosin
-reduce inhibition of tropomyosin upon the myofilaments
-cause depolarization of muscle
thanks
2) greater overjet of anterior teeth then::
-shorter the cusps of posterior teeth
-greater intercondylar distance
-lesser intercondylar distance
-more acute benett angle
-more obtuse benett angle
3)internal carotid arteries become occluded then blood reach arterial circle via:
-vertebral artery???
4) role of calcium in activation of cardiac muscle
-couple myosin n actin to form actinomyosin
-reduce inhibition of tropomyosin upon the myofilaments
-cause depolarization of muscle
thanks
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i think u were wrong on the last Q..
Ca2+ binf tropnin c which inturn cause the removal of tropomyosin from actin.. this is seen in skeletal and cardaic mm. im smooth ca2+ will bind calmdoulin-->activte kinase-->phosop of myosin light chain.. thats my opinion..lets here some other guys.. 🙂
