Lateral hip. L4?be specific about what part of the "hip" - anterior, lateral, posterior/buttock?
Lateral hip. L4?be specific about what part of the "hip" - anterior, lateral, posterior/buttock?
be specific about what part of the "hip" - anterior, lateral, posterior/buttock?
I guess you could say that certain lumbar dermatomes cross the region of the lateral hip.....I’m assuming you’re gonna say: posterior L5 or S1, lateral L4, and anterior L3? TIA!
Still nonspecific unless it is the occasional classic full spectrum of the dermatome/myotome. Any level can go to the buttock or lateral hip. L2, 3, 4 could be anywhere in the anterior or lateral thigh…. Or just glute.I guess you could say that certain lumbar dermatomes cross the region of the lateral hip.....
Appreciate all the input so far.Still nonspecific unless it is the occasional classic full spectrum of the dermatome/myotome. Any level can go to the buttock or lateral hip. L2, 3, 4 could be anywhere in the anterior or lateral thigh…. Or just glute.
NO it can’tSacral tarlov cysts? Could explain pain.
lol not falling for thisNO it can’t
Tarlov cysts do not hurt.lol not falling for this
Prove itTarlov cysts do not hurt.
How?Prove it
Thank youHow?
How do you account for the 174 who declined treatments from a statistical standpoint? BOCF.I do think it's the cyst. If all of it doesn't help, ask an IR to do a cyst aspiration and injection of fibrin. That's a right S2 cyst correct?
but a perineural cyst is different than a tarlov cyst right?How do you account for the 174 who declined treatments from a statistical standpoint? BOCF.
View attachment 369855
I don't think so for this study. Will have to relook.but a perineural cyst is different than a tarlov cyst right?
I’ve had at least one patient in last year that I’ve narrowed down to this. Buttock pain only.How?
I've never heard of Tarlov cysts causing pain.
How exactly did you do that?I’ve had at least one patient in last year that I’ve narrowed down to this. Buttock pain only.
Honestly I think they’re like anything else in the spine, annular tears, disc protrusions, spondylosis. In the right setting and with enough inflammation they can be painful but typically are asymptomaticHow exactly did you do that?
This discussion has come up repeatedly. I was always taught they don't hurt. Truthfully, I don't know why they would hurt either.
It's pain, not diabetes.
In the right setting and with enough inflammation? What are you even talking about? The right setting is never.Honestly I think they’re like anything else in the spine, annular tears, disc protrusions, spondylosis. In the right setting and with enough inflammation they can be painful but typically are asymptomatic
Tbone you’ve established your ignorance before. No need to explain buddyIn the right setting and with enough inflammation? What are you even talking about? The right setting is never.
I could be convinced cysts cause symptoms of IR drains a cyst and it relieves their symptoms. But excluding other causes doesn’t convince me the cysts are symptomatic, plenty of people have pain symptoms without a clear cause.In the right setting and with enough inflammation? What are you even talking about? The right setting is never.
Cmon @clubdeac relax a little bit. If you want to start a new thread called “ the ignorance of Tboned” go ahead. But re: this thread, I’m always willing to learn. Explain what you’re talking about, I’m interested to hear your thoughts on the inflammatory component or a Tarlov cyst. How are you determining that? It’s not an apples to apples comparison with annular tears and other pathology you hinted at.Tbone you’ve established your ignorance before. No need to explain buddy
I think of that as "Patient Selection"How do you account for the 174 who declined treatments from a statistical standpoint? BOCF.
208 had cysts and were the selected patients. 34 had direct surgery referral and 174 said no thanks. Lets say every one of those 208 was treated and had excellent results. Now lets say all 208 had terrible results. It would completely change the outcomes. They only treated 213.I don't think of Tarlov cysts as necessarily inflammatory. There was some early postulations about inflammation playing a role in their creation, but I don't know if they really cause inflammation. I do see that they are capable of generating a great deal of pressure. As you look for them more, you'll see cysts that cause remodeling of the bone around them from outward pressure. It's not unreasonable to me then that the pressure causes a neuropraxia of a nerve root inside or outside the cyst. Drainage by IR or an aggressive pain doc allows one to verify that this is the generator. That fibrin injection thing may work sometimes, but I've seen mixed results. I haven't seen a surgeon willing to work on them.
I think of that as "Patient Selection"
I'm not sure I get the BOCF? Google says baseline-observation-carried-forward which might make sense if you're looking at this like a RCT for ITT, but this is a retrospective case series.
I interpret this as a subgroup they selected for have good results with drainage/fibrin patching, but they may have weeded out a lot of cases that they didn't think would have good results. I would love to hear more about that population too and understand the selection criteria.
So we know inflammation is a necessary component to experience pain in spinal stenosis, facet arthritis, discogenic lbp and particularly radicular pain. You need two things to experience pain in these scenarios, some structural or anatomical pathology AND inflammation. If you take one of these away, the patient typically won’t experience pain regardless of the MRI findings. There are studies showing this.Cmon @clubdeac relax a little bit. If you want to start a new thread called “ the ignorance of Tboned” go ahead. But re: this thread, I’m always willing to learn. Explain what you’re talking about, I’m interested to hear your thoughts on the inflammatory component or a Tarlov cyst. How are you determining that? It’s not an apples to apples comparison with annular tears and other pathology you hinted at.
I’m talking about typical spinal pain, not CRPS, central pain syndrome, fibromyalgia, phantom limb pain, chronic pelvic pain etc.Pain does not require an anatomical basis.
Me tooI’m talking about typical spinal pain, not CRPS, central pain syndrome, fibromyalgia, phantom limb pain, chronic pelvic pain etc.
technically, you do not require anything anatomic to experience pain. see fibromyalgia (at least based on what we know about it now)So we know inflammation is a necessary component to experience pain in spinal stenosis, facet arthritis, discogenic lbp and particularly radicular pain. You need two things to experience pain, some structural or anatomical pathology AND inflammation. If you take one off these away, the patient typically won’t experience pain regardless of the MRI findings. There are studies showing this.
I’ve seen multiple patients with asymptomatic tarlov cysts as well those where nothing else explains their pain other than the tarlov cyst. In those cases I presume the cyst is pressing on pain generating structures. However based on what we know about spinal pathology, there is likely an inflammatory component. I don’t know that but am rather extrapolating from what we know about other spinal pathologies
I guess no one read what I wrote. Oh welltechnically, you do not require anything anatomic to experience pain. see fibromyalgia (at least based on what we know about it now)
pain is a subjective experience.
and when i look at MRI scans for failed back syndrome, im not seeing a lot of inflammation. even facet arthropathy - is that were purely inflammation, then steroidal therapy would be the mainstay and only treatment, right?
Studies are from fellowship as are the text book references. I could try and find them but this is like finding the study that shows seat belts save lives, not really necessary. Again I’m talking about the 95% of scenarios with lumbar radic, spinal stenosis, facet arthritis etcyes we did.
so why do you feel that inflammation is a necessary component to experience pain from the 4 conditions you state?
you state there are studies. can you post them?
I’m not conflating anything. But yes that’s exactly what I’m talking about. Those are inflammatory mediators and although they may not directly raise ESR or CRP, that doesn’t mean there isn’t inflammation present. I mean cmon, we all prescribe NSAIDs for inflammation everyday. Most all of these patients have normal ESRs. Does that mean we’re not treating inflammation or we’re conflating something? This really shouldn’t be difficult or controversialhow often do we see edema or elevated ESR in these patients with arthritic spines?
then again, maybe you are conflating the presence of inflammatory cytokines - substance P, histamines - in the chemical milieu contributing to neuropathic pain in your definition of inflammation....
I give up with you. Steroid injections work remarkably well in the short term (~3 months) for spinal stenosis and facet arthritis. You must be doing them wrong.inflammatory mediators =/= inflammation.
huge difference.
which is why steroid injections dont work for facet arthritis or degenerative spinal stenosis with neurogenic claudication.
and ive stopped prescribing NSAID for the types of conditions you describe.
why?
they dont work.
because it is not inflammation that is driving their pain.
Steroid injections do not work for facet arthritis.I give up with you. Steroid injections work remarkably well in the short term (~3 months) for spinal stenosis and facet arthritis. You must be doing them wrong.
I'd be interested in hearing your theory regarding the pathophysiology of facet mediated pain, spinal stenosis, discogenic lbp, painful radiculopathy etc. What is your understanding of nociception?
And when I refer to inflammation I do mean substance P, IL-6, cytokines, TNF-alpha, PLA2 and on and on...