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- Nov 20, 2001
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I just recently started work as a private practice attending and found Jet's comments dead on.
I'd like to suggest that the newbies get really handy with LMAs. Where I trained, GETA was the standard and the use of LMAs were rare. See, all of my attendings were deathly scared of the nurses and the residents not keeping the patients deep enough, and therefore didn't trust us with unsecured airways. It's a lot easier to check your stocks and watch TV when the patient is tubed and paralyzed when the CA-1 is in the room doing that ORIF of the forearm.
Now I often have schedules in which I'm doing 10 or so knee scopes a day. Each one is roughly 15-20 mins long from skin to skin and it's strictly LMAs and deep removal. More often than not,I sometimes even place the patient on pressure controlled ventilation.
As a resident at the ivory tower of academics, the standard technique was to hit the patient with 2-2.5mg/kg of propofol, a cc of fentanyl, and then mask for a minute or so with sevo before placing the fully deflated LMA. Later, I and few others became proponents of the technique introduced to us by attendings who had come out of private practice that involved simply slugging the patient with propofol (usually 4mg/kg) to cause apnea, throw in the LMA partially inflated, and then hook up the patient to a circuit and wait for spontaneous ventilation to resume. Without an opioid, the patient usually resumes breathing within 1-2 minutes. I then cranked up the Sevo dial to 6-8% with good flows and allow the patient to deepen himself while prepping occurs. N2O +/-
Unfortunately, at my surgery centers, things move really fast and really efficiently. The nurses and the orthopods move in like vultures once the LMA is in and even with the stimulus of positioning, I used to see some sort of myoclonic movement (often in young men) 1-2 minutes later if I didn't quickly hit the patient with another 1-2 mg/kg more of propofol shortly after LMA placement.
The solution, in large part, has been to put patients on the vent, watching for peak inspiratory pressures greater than 20mm. The patients get a good hit of gas thus preventing the spasmic motions and movement at incision, the propofol can be avoided, the patient stays oxygenated, and by avoiding opioids, I can get them back breathing shortly after the case is started. Myoclonus still (rarely) occurs, but that's why I have extra propofol handy.
I'd like to suggest that the newbies get really handy with LMAs. Where I trained, GETA was the standard and the use of LMAs were rare. See, all of my attendings were deathly scared of the nurses and the residents not keeping the patients deep enough, and therefore didn't trust us with unsecured airways. It's a lot easier to check your stocks and watch TV when the patient is tubed and paralyzed when the CA-1 is in the room doing that ORIF of the forearm.
Now I often have schedules in which I'm doing 10 or so knee scopes a day. Each one is roughly 15-20 mins long from skin to skin and it's strictly LMAs and deep removal. More often than not,I sometimes even place the patient on pressure controlled ventilation.
As a resident at the ivory tower of academics, the standard technique was to hit the patient with 2-2.5mg/kg of propofol, a cc of fentanyl, and then mask for a minute or so with sevo before placing the fully deflated LMA. Later, I and few others became proponents of the technique introduced to us by attendings who had come out of private practice that involved simply slugging the patient with propofol (usually 4mg/kg) to cause apnea, throw in the LMA partially inflated, and then hook up the patient to a circuit and wait for spontaneous ventilation to resume. Without an opioid, the patient usually resumes breathing within 1-2 minutes. I then cranked up the Sevo dial to 6-8% with good flows and allow the patient to deepen himself while prepping occurs. N2O +/-
Unfortunately, at my surgery centers, things move really fast and really efficiently. The nurses and the orthopods move in like vultures once the LMA is in and even with the stimulus of positioning, I used to see some sort of myoclonic movement (often in young men) 1-2 minutes later if I didn't quickly hit the patient with another 1-2 mg/kg more of propofol shortly after LMA placement.
The solution, in large part, has been to put patients on the vent, watching for peak inspiratory pressures greater than 20mm. The patients get a good hit of gas thus preventing the spasmic motions and movement at incision, the propofol can be avoided, the patient stays oxygenated, and by avoiding opioids, I can get them back breathing shortly after the case is started. Myoclonus still (rarely) occurs, but that's why I have extra propofol handy.