USMLE images
Started by Zuhal
what other defect do you see with lithium as a teratogen?
Advertisement - Members don't see this ad
Much thx
I dont know a good source for ekgs but i got some lectures of one indian guy who gives lectures on it and its quite good for step 2 i guess
1. what hormone drives this tumor
2. which other neoplasms produce a similar histology finding
1. TSH?
2. Serous papillary cystadenoma of the ovary, Meningioma, Mesothelioma.
this thread is so sexy!
Lady comes in with pain when she eats. Decide to do a blood smear and see this. Dx?
Looks like spherocytes
Advertisement - Members don't see this ad
Rouleaux? can be elevated protein or ESR, so...temporal arteritis?
Pain when she eats might be jaw claudication?
Agreed. Jaw caludication=temporal arteritis= ESR increase
Just wondering, how were you guys able to associate rouleaux with high ESR?
In first aid, I only see it mentioned one time in the end section where it just talks about high yield points.
Did y'all just remember it from class or something?
In first aid, I only see it mentioned one time in the end section where it just talks about high yield points.
Did y'all just remember it from class or something?
General pathology section of FA
alpha3 chain of collagen iv?
EDIT: wow nevermind at my last edit, i hope no one saw that lol
Advertisement - Members don't see this ad
yeah i knew that temporal arteritis causes increased ESR.
i guess my understanding isn't enough to automatically associate a high erythrocyte sedimentation rate with rouleaux formation haha
Anytime we have inflammation, the liver makes a lot of fibrinogen and other "acute phase reactants". This fibrinogen is positively charged so when it goes out into the bloodstream and mixes with the negatively charged RBC, it causes them to clump around it and we get that stacked rouleaux appearance.
Are you thinking Alport Syndrome? I don't think they have any lung involvement. It's mostly kidneys, ears and eyes.
Just think about what ESR actually means..... rate of RBC settling at the bottom of a tube; when rouleaux occurs.. the RBCs stick together and becoming heavier... and more will sink faster to the bottom of the tube, ala higher sedimentation rate.
Good point!
No he doesn't have Goodpastures, not with this type of MRI. Another hint: He has sinusitis and otitis media.
kinda and kinda not...need a bit more info on the question. keep in mind goodpasture's also does affect the lungs, specifically the alveoli. it can be antibodies against the basement membrane or getting a bit more info it could be antibodies against neutrophils.
which one is it brah?
Advertisement - Members don't see this ad
Yes it is Wegners and yes they have c-ANCA antibodies, you're almost there.
Against which specific cellular protein/structure are these antibodies made?
nice job dude...keep it up. 👍
next Q for the class...
1. what is this condition associated with, with respect to that organ?
2. causes of this in adults?
Is that a pseudocyst?
GET SMASHED
oh lol... it's proteinase 3 (i just added this to my first aid yesterday from a uworld question).
Although this question isn't hinting at pauci-immune mediated cresentic GN.. in that case, it would be P-ANCA (anti-myeloperoxidase)...I cheated on this one..
Right but what is it about struge weber that makes the patient have an increase in preload and a decrease in afterload?
Decreased cross sectional area due to arterioles being non-compliant?
I'm horrible with vasculitis/heart so this is a complete shot in the dark
AVM associated with Sturge weber -> increased preload. Not sure what exactly causes reduced after load though.
Advertisement - Members don't see this ad
