Official NBDE Part 1 Study Q & A Thread
Started by tinman831
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I think you are the one who asked us about how blood pressure increases with vasoconstriction, right?
Why did BP increase during vasoconstriction? you have the same amount of volume but less space. That's how you can increase the pressure. Another way to increase pressure is the same space but increased volume.
In this case, you have the same amount of blood in ventricle. This is a closed container cuz all the valves are closed. And if u contract the ventricle, the space gets smaller while the volume stays the same. That's why the pressure increases.
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during active eruption ,bone formation is often seen at the base of the socket .this newly formed bone is usually in form of
1osteons
2compact bone
radiating trabeculae
horizontal trabeculae ....answer
could'nt find anything relevant for horizontal trabeculae,does anyone have any idea what these r?
1osteons
2compact bone
radiating trabeculae
horizontal trabeculae ....answer
could'nt find anything relevant for horizontal trabeculae,does anyone have any idea what these r?
My exam is n 10 days.
I'm not ready - can not grasp the immensity.
I'm studying more and getting confused more.
I'm getting in ASDA - 75-80 in first time, in second time - much more (about 95 - just remember answers).
There are more than 4000 Q in ASDA papers. Does in make any sense to read them all? How useful they are for the real exam?
hey Hatico...are u doing twice all the ASDA papers or just some of them...did u buy tht 'crack the NBDE'...iam also almost done once with Asda and scoring around 75-80..but still not confident...my exam s exactly 1 month 1 day from today....
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don't buy Crack. It's so different from the actual boards and has tons of errors. Who would've thought that working condyle moves downward, forward and medially? Even high school kids would make better study aid than that.
I have checked Crack's review - mostly negative. There is even more errors than in the Decks?
Koala, please tell us what do you think about ASDA papers?
Difference between Crack NBDE and the real NBDE = 10 miles apart.
Difference between ASDA paper and the real NBDE = 1 inch apart.
I came home and read dental decks again. I found out that I missed alot of things that dental deck said important or note. Don't miss those information. I think whoever took the boards remembered questions from the actual board exam and wrote it under 'important' and 'note'.
There were about 10 repeated questions from old ASDA paper.
All the questions were similiar to the released ASDA paper, but obviously the questions were different.
ya i know , adenosine, lactic acid and co2 are the byproducts of metabolism but when they are released they act on arterioles and arteries to vasodilate ,decrease vascular resistance, increase blood flow....but not on capillaries..
No pookan...it happens immediately after a refractory period.
Negative after potential-stimulus which has lower intensity than the normal will cause a change.
Positive after potential-it will take more than a normal intensity stimulus to cause a change.
http://books.google.com/books?id=mP...DQQ6AEwAg#v=onepage&q=spike potential&f=false
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ok..so it means tht
1)spike potential occurs i.e., phase of depolarization no stimulus can be initiated hence absolute refractory period..thn
2)-ve after potential occurs ....its also part of action potential as Na+ ions are still present inside the cell then
3) +ve after potential occurs where Na-K pump comes and takes out Na+ out of cell and brings K+ inside...here greater intensity must be provided to initiate another potential i.e.,refractory period..
Am i in right direction? plz correct me where ever iam wrong...thank u
What processes determines the size of the mouth??
Which processes form the face??
lines of owen in dentin are comparable to which in emamel???
what forms the hertwig epithlial root sheet???
The problem cystic fibrosis is inabilit of sweat gland to absoorb which??? Na and Cl
The S shape of odontoblastic processes is due to what factor?
Which of te following acid does not cause dental caries?? acetic, oleic, proic and i cant remember d last option
there is no evidence that insipidus causes kidney stone but UTI causes kidney stone cos the microbes destroy the citrate producing cell. citrate in the kidney helps prevent calcium stone formation.
Which processes form the face??
lines of owen in dentin are comparable to which in emamel???
what forms the hertwig epithlial root sheet???
The problem cystic fibrosis is inabilit of sweat gland to absoorb which??? Na and Cl
The S shape of odontoblastic processes is due to what factor?
Which of te following acid does not cause dental caries?? acetic, oleic, proic and i cant remember d last option
there is no evidence that insipidus causes kidney stone but UTI causes kidney stone cos the microbes destroy the citrate producing cell. citrate in the kidney helps prevent calcium stone formation.
Antifungal agents are specific for fungal and not bacteria because of which of the following
A) Bacteria cell wall is thicker than fungal
A) Bateria is prokaryote while fungal is Eukaryote
C) Cell wall of fungus contains mycolic acid which is where the antifungal agent binds/ is specific to/for
D) Bacteria contain lipopolysaccharides while fungus contain proteoglycan ans
y do u think its D ...antifungal agents acts on ergosterol...its a sterol not a proteoglycan..
A) Bacteria cell wall is thicker than fungal
A) Bateria is prokaryote while fungal is Eukaryote
C) Cell wall of fungus contains mycolic acid which is where the antifungal agent binds/ is specific to/for
D) Bacteria contain lipopolysaccharides while fungus contain proteoglycan ans
y do u think its D ...antifungal agents acts on ergosterol...its a sterol not a proteoglycan..
It can be only D (closest to truth). Mycobacteria and Actinomycetes contain mycolic acid - no fungi.
Ergosterol is component of fungal cell membrane, polisaccharide - cell wall. But no proteoglycans.
NOt all antifungal agents act on ergosterol - only clotrimazole and polienic antibiotics. Other acts on cytochromes.
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Originally Posted by ionomer
lines of owen in dentin are comparable to which in emamel???
lines of reitzus
No, it's neonatal line. ( Lines of Reitzius on enamel - Von Ebner's lines in dentin)
Hatico, i would say have agreed with you but when i researched it, i got it from wikipedia, except this particular article is wrong from wikipedia. Here is the link http://en.wikipedia.org/wiki/Striae_of_Retzius. Read it under the subtopic "Qualities"🙂
IgA antibody is the first line of defense against infections at the
mucous membrane. It is usually an early specific antibody. Which of the
following statements regarding IgA is not true?
a. Complement fixation tests for IgA antibody will be positive if specific IgA anti-
body is present
b. IgA is not found in saliva, therefore an IgA diagnostic test on saliva would have
no value
c. IgA can be destroyed by bacterial proteases
d. IgA is absent in colostrum
e. IgA is a small molecule with a molecular weight of 30,000 kDa
mucous membrane. It is usually an early specific antibody. Which of the
following statements regarding IgA is not true?
a. Complement fixation tests for IgA antibody will be positive if specific IgA anti-
body is present
b. IgA is not found in saliva, therefore an IgA diagnostic test on saliva would have
no value
c. IgA can be destroyed by bacterial proteases
d. IgA is absent in colostrum
e. IgA is a small molecule with a molecular weight of 30,000 kDa
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