Official NBDE Part 1 Study Q & A Thread

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Please post all study questions/answers for the NBDE Part 1 in this thread. Good luck!

As a side note, this is not the place for sales ads. Discussion of remembered questions appearing on the exam is also not permitted.
 
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Hatico said:
During the period of isovolumetric contraction, which of the following happens?

  1. The semilunar valves are open.
  2. The left ventricular pressure is rising rapidly.ans
  3. The aortic pressure is slightly less than the left ventricular pressure.
  4. The right ventricular pressure is greater than the left ventricular pressure.
I see that A,C,D are not correct. Why is B correct? There is all valves are closed - how can ventricular pressure be "rising rapidly"? Should be left atria? Please, explain!
Click to expand...

I think you are the one who asked us about how blood pressure increases with vasoconstriction, right?

Why did BP increase during vasoconstriction? you have the same amount of volume but less space. That's how you can increase the pressure. Another way to increase pressure is the same space but increased volume.

In this case, you have the same amount of blood in ventricle. This is a closed container cuz all the valves are closed. And if u contract the ventricle, the space gets smaller while the volume stays the same. That's why the pressure increases.
 
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I did never ask about BP and vasoconstriction.
Thank you for explanation about ventricular pressure:bow:
 
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during active eruption ,bone formation is often seen at the base of the socket .this newly formed bone is usually in form of
1osteons
2compact bone
radiating trabeculae
horizontal trabeculae ....answer

could'nt find anything relevant for horizontal trabeculae,does anyone have any idea what these r?
 
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Holly J said:
@Hatico....what date is your exam? Ready for it...How much are you getting on asda and other papers?
Click to expand...

My exam is n 10 days.
I'm not ready - can not grasp the immensity.
I'm studying more and getting confused more.
I'm getting in ASDA - 75-80 in first time, in second time - much more (about 95 - just remember answers).
There are more than 4000 Q in ASDA papers. Does in make any sense to read them all? How useful they are for the real exam?
 
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Hatico said:
My exam is n 10 days.
I'm not ready - can not grasp the immensity.
I'm studying more and getting confused more.
I'm getting in ASDA - 75-80 in first time, in second time - much more (about 95 - just remember answers).
There are more than 4000 Q in ASDA papers. Does in make any sense to read them all? How useful they are for the real exam?
Click to expand...
Well honestly it just gives you an idea about the exam...and if you are lucky enough some repeats too. It will be helpful no matter what.
 
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PooKan said:
during active eruption ,bone formation is often seen at the base of the socket .this newly formed bone is usually in form of
1osteons
2compact bone
radiating trabeculae
horizontal trabeculae ....answer

could'nt find anything relevant for horizontal trabeculae,does anyone have any idea what these r?
Click to expand...

Direction of trabecules depend on forces. If tooth is pushing out - there is horisontal force. I think if tooth is pulling into the alveola - radial force.
 
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PooKan said:
during active eruption ,bone formation is often seen at the base of the socket .this newly formed bone is usually in form of
1osteons
2compact bone
radiating trabeculae
horizontal trabeculae ....answer

could'nt find anything relevant for horizontal trabeculae,does anyone have any idea what these r?
Click to expand...
This is all I could get right now:
Horizontal trabeculae are seen in places where there is no load,they just add to the bulk...Vertical/radiating trabeculae are seen in areas of high load.
 
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Does anyone have pattern for height of contour?
In my pattern lingual height of contour on mandibular P,M is on medial third. But ASDA says it's on occlusal third on 2P.
 
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Hatico said:
Does anyone have pattern for height of contour?
In my pattern lingual height of contour on mandibular P,M is on medial third. But ASDA says it's on occlusal third on 2P.
Click to expand...
i think its middle 3rd..look into pg number- 623 of first aid...they have given it very nicely
 
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Hatico said:
My exam is n 10 days.
I'm not ready - can not grasp the immensity.
I'm studying more and getting confused more.
I'm getting in ASDA - 75-80 in first time, in second time - much more (about 95 - just remember answers).
There are more than 4000 Q in ASDA papers. Does in make any sense to read them all? How useful they are for the real exam?
Click to expand...
hey Hatico...are u doing twice all the ASDA papers or just some of them...did u buy tht 'crack the NBDE'...iam also almost done once with Asda and scoring around 75-80..but still not confident...my exam s exactly 1 month 1 day from today....
 
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PooKan said:
hey Hatico...are u doing twice all the ASDA papers or just some of them...did u buy tht 'crack the NBDE'...iam also almost done once with Asda and scoring around 75-80..but still not confident...my exam s exactly 1 month 1 day from today....
Click to expand...
I'm doing twice not all of them - only not answered correctly, confusing or 50/50 choice.
I did not buy Crack.
 
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Hatico said:
I'm doing twice not all of them - only not answered correctly, confusing or 50/50 choice.
I did not buy Crack.
Click to expand...
don't buy Crack. It's so different from the actual boards and has tons of errors. Who would've thought that working condyle moves downward, forward and medially? Even high school kids would make better study aid than that.
 
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PooKan said:
capillary diameter directly influenced by byproducts of metabolism...wht are they?
Click to expand...

pookan...I saw this question before...Its Adenosine-byproduct of metabolism. I am trying to find the exact mechanism here. It seems it is deposited on the vessel wall.
 
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Hatico said:
I have checked Crack's review - mostly negative. There is even more errors than in the Decks? :laugh:
Koala, please tell us what do you think about ASDA papers?
Click to expand...

Difference between Crack NBDE and the real NBDE = 10 miles apart.
Difference between ASDA paper and the real NBDE = 1 inch apart.

I came home and read dental decks again. I found out that I missed alot of things that dental deck said important or note. Don't miss those information. I think whoever took the boards remembered questions from the actual board exam and wrote it under 'important' and 'note'.

There were about 10 repeated questions from old ASDA paper.
All the questions were similiar to the released ASDA paper, but obviously the questions were different.
 
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Holly J said:
pookan...I saw this question before...Its Adenosine-byproduct of metabolism. I am trying to find the exact mechanism here. It seems it is deposited on the vessel wall.
Click to expand...
ya i know , adenosine, lactic acid and co2 are the byproducts of metabolism but when they are released they act on arterioles and arteries to vasodilate ,decrease vascular resistance, increase blood flow....but not on capillaries..
 
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PooKan said:
ya i know , adenosine, lactic acid and co2 are the byproducts of metabolism but when they are released they act on arterioles and arteries to vasodilate ,decrease vascular resistance, increase blood flow....but not on capillaries..
Click to expand...

Wiki: Localized tissues utilize multiple ways to increase blood flow including releasing vasodilators, primarily adenosine, into the local instersitial fluid which diffuses to capillary beds provoking local vasodilation.
 
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Holly J said:
Wiki: Localized tissues utilize multiple ways to increase blood flow including releasing vasodilators, primarily adenosine, into the local instersitial fluid which diffuses to capillary beds provoking local vasodilation.
Click to expand...
ok...but still one doubt left...metabolites diffuses into capillary beds but vasodilation and resistance s due to arterioles not capillaries..
 
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subliminal fringe of motor neuron pool is useful concept in explaining central facilitation...
plz explain this...i tried reading from previous related posts but couldnt understand..thankz
 
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Holly J said:
No pookan...it happens immediately after a refractory period.
Negative after potential-stimulus which has lower intensity than the normal will cause a change.
Positive after potential-it will take more than a normal intensity stimulus to cause a change.
Click to expand...
iam sorry could u explain me again with more detail...i know now it happens after refractory period..but after absolute refractory period ,relative refractory period comes right?
 
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PooKan said:
iam sorry could u explain me again with more detail...i know now it happens after refractory period..but after absolute refractory period ,relative refractory period comes right?
Click to expand...
ok..so it means tht
1)spike potential occurs i.e., phase of depolarization no stimulus can be initiated hence absolute refractory period..thn
2)-ve after potential occurs ....its also part of action potential as Na+ ions are still present inside the cell then
3) +ve after potential occurs where Na-K pump comes and takes out Na+ out of cell and brings K+ inside...here greater intensity must be provided to initiate another potential i.e.,refractory period..
Am i in right direction? plz correct me where ever iam wrong...thank u
 
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What processes determines the size of the mouth??

Which processes form the face??

lines of owen in dentin are comparable to which in emamel???

what forms the hertwig epithlial root sheet???

The problem cystic fibrosis is inabilit of sweat gland to absoorb which??? Na and Cl

The S shape of odontoblastic processes is due to what factor?

Which of te following acid does not cause dental caries?? acetic, oleic, proic and i cant remember d last option


there is no evidence that insipidus causes kidney stone but UTI causes kidney stone cos the microbes destroy the citrate producing cell. citrate in the kidney helps prevent calcium stone formation.
 
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Antifungal agents are specific for fungal and not bacteria because of which of the following

A) Bacteria cell wall is thicker than fungal
A) Bateria is prokaryote while fungal is Eukaryote
C) Cell wall of fungus contains mycolic acid which is where the antifungal agent binds/ is specific to/for
D) Bacteria contain lipopolysaccharides while fungus contain proteoglycan ans

y do u think its D ...antifungal agents acts on ergosterol...its a sterol not a proteoglycan..
 
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ionomer said:
What processes determines the size of the mouth??

Which processes form the face??

lines of owen in dentin are comparable to which in emamel???
perinatal line?
what forms the hertwig epithlial root sheet???
outer and inner enamel epithelium

The problem cystic fibrosis is inabilit of sweat gland to absoorb which??? Na and Cl
Cl-
The S shape of odontoblastic processes is due to what factor?
odontoblastic overpopulation

Which of te following acid does not cause dental caries?? acetic, oleic, proic and i cant remember d last option


there is no evidence that insipidus causes kidney stone but UTI causes kidney stone cos the microbes destroy the citrate producing cell. citrate in the kidney helps prevent calcium stone formation.
Click to expand...

Do you have Decks? I believe all answers are there.
 
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PooKan said:
Antifungal agents are specific for fungal and not bacteria because of which of the following

A) Bacteria cell wall is thicker than fungal
A) Bateria is prokaryote while fungal is Eukaryote
C) Cell wall of fungus contains mycolic acid which is where the antifungal agent binds/ is specific to/for
D) Bacteria contain lipopolysaccharides while fungus contain proteoglycan ans

y do u think its D ...antifungal agents acts on ergosterol...its a sterol not a proteoglycan..
Click to expand...

It can be only D (closest to truth). Mycobacteria and Actinomycetes contain mycolic acid - no fungi.
Ergosterol is component of fungal cell membrane, polisaccharide - cell wall. But no proteoglycans.
NOt all antifungal agents act on ergosterol - only clotrimazole and polienic antibiotics. Other acts on cytochromes.
 
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Hatico said:
1)Triglyceride absorbed into the lymphatic system is transported to the liver as which of the following?

  1. Very low density lipoprotein
  2. Low density lipoprotein
  3. Chylomicrons ans
  4. Liposomes
  5. Micelles
This Q is confusing.
If answer is 3 that Q is wrong! Chylomicrons are never transported to the liver - only to extrahepatic tissues.
If Q is correct that answer is wrong. Only LDL and HDL carry tryglicerids (and cholesterol) to the liver
Click to expand...
Chylomicrons-carry TGs to extrahepatic tissues.
IDL-carry TGs back to liver.
HDL-doesnt carry TGs...It carries only cholesterol back to the liver(reverse chelesterol transport)
 
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Yes. HDL carry cholesterol to the liver.
LDL - cholesterol and TG to the liver.
CHM and VLDL - to extrahepatic tissues
Answr 2 is correct🙄
 
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ionomer said:
What processes determines the size of the mouth?? max and mandibular processes

Which processes form the face??

lines of owen in dentin are comparable to which in emamel???
lines of reitzus
what forms the hertwig epithlial root sheet??? Cervical loop forms HERS

The problem cystic fibrosis is inabilit of sweat gland to absoorb which??? Na and Cl

The S shape of odontoblastic processes is due to what factor? Crowding of the odntoblastic processes

Which of te following acid does not cause dental caries?? acetic, oleic, proic and i cant remember d last option


there is no evidence that insipidus causes kidney stone but UTI causes kidney stone cos the microbes destroy the citrate producing cell. citrate in the kidney helps prevent calcium stone formation.
Click to expand...


Them them up guys
 
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Hatico said:
Yes. HDL carry cholesterol to the liver.
LDL - cholesterol and TG to the liver.
CHM and VLDL - to extrahepatic tissues
Answr 2 is correct🙄
Click to expand...


Chylomicron transports dietry triglyceride from the GIT to muscle, adipose tissues and LIVER. it contains 85%-92% of triglyceride

LDL is the primary carrier of Cholesterol to the liver.

So i will go with answer 3
 
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hey guys thrombotic occlusion of coronary artery results in (all 3 options are right)
-infraction of myocardium
-fibrosis of myocardium and
-no changes in myocardium
how is 3 rd option also right?
 
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PooKan said:
hey guys thrombotic occlusion of coronary artery results in (all 3 options are right)
-infraction of myocardium
-fibrosis of myocardium and
-no changes in myocardium
how is 3 rd option also right?
Click to expand...

via arterial collaterales
 
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Originally Posted by ionomer

lines of owen in dentin are comparable to which in emamel???
lines of reitzus


No, it's neonatal line. ( Lines of Reitzius on enamel - Von Ebner's lines in dentin)

 
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Hatico said:
Originally Posted by ionomer

lines of owen in dentin are comparable to which in emamel???
lines of reitzus


No, it's neonatal line. ( Lines of Reitzius on enamel - Von Ebner's lines in dentin)
Click to expand...

Hatico, i would say have agreed with you but when i researched it, i got it from wikipedia, except this particular article is wrong from wikipedia. Here is the link http://en.wikipedia.org/wiki/Striae_of_Retzius. Read it under the subtopic "Qualities"🙂
 
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IgA antibody is the first line of defense against infections at the
mucous membrane. It is usually an early specific antibody. Which of the
following statements regarding IgA is not true?
a. Complement fixation tests for IgA antibody will be positive if specific IgA anti-
body is present
b. IgA is not found in saliva, therefore an IgA diagnostic test on saliva would have
no value
c. IgA can be destroyed by bacterial proteases
d. IgA is absent in colostrum
e. IgA is a small molecule with a molecular weight of 30,000 kDa
 
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System32 said:
IgA antibody is the first line of defense against infections at the
mucous membrane. It is usually an early specific antibody. Which of the
following statements regarding IgA is not true?
a. Complement fixation tests for IgA antibody will be positive if specific IgA anti-
body is present
b. IgA is not found in saliva, therefore an IgA diagnostic test on saliva would have
no value
c. IgA can be destroyed by bacterial proteases
d. IgA is absent in colostrum
e. IgA is a small molecule with a molecular weight of 30,000 kDa
Click to expand...

IgA is present in colostrum so option d id the correct one
 
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System32 said:
IgA antibody is the first line of defense against infections at the
mucous membrane. It is usually an early specific antibody. Which of the
following statements regarding IgA is not true?
a. Complement fixation tests for IgA antibody will be positive if specific IgA anti-
body is present IgA activates complement very weak, but possible
b. IgA is not found in saliva, therefore an IgA diagnostic test on saliva would have
no value IgA is found in saliva predominantly
c. IgA can be destroyed by bacterial proteases yes
d. IgA is absent in colostrum not correct
e. IgA is a small molecule with a molecular weight of 30,000 kDa too much for IgA
Click to expand...

:idea:
 
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PooKan said:
hey guys thrombotic occlusion of coronary artery results in (all 3 options are right)
-infraction of myocardium
-fibrosis of myocardium and
-no changes in myocardium
how is 3 rd option also right?
Click to expand...
Is the question complete?
Either its 1 and 2 or just 3! If the collaterals are present then there wouldnt be infarction/fibrosis of myocardium(1 &2).
What was the answer Pookan?
Hatico....mind explaining your answer?
 
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