Smother it in peanut butter? Wrap it in a piece of lunch meat? Idk that’s all I’ve got.
Not gonna lie, I'm seriously considering melting some chocolate and dunking it in it before my next dose.
Do itttttt
And owners always think I’m just being over dramatic about the taste! No, you cannot crush that pill and mix it with food, or your cat will never want to eat again.
Bonus points for (at least the human dose) being a giant pill, feels like every time I take it, it goes down sideways and prolongs the bitterness.
I mean, I developed a severe aversion to the beverage I used to take it when I was on it as kid so... I hear ya, cats.
The tiny tabs I use with cats are coated but only 50mg. So much easier to give as a coated tablet.
Based on my experience tonight, the next week is going to be much more pleasant with the gelatin capsules. Thanks again! Metro is gross.
I use pill pockets for Cindy and for a week or two she'll just eat them, and then she'll decide nah, hate these...so I'll switch flavors. And then she'll start eating them again. Rinse and repeat...she is not the brightest bulb
That's fine, I'm not trying to make them like pill pockets. I'm just doing the same thing as a gelatin capsule and masking the grossness with a thin layer.
She's special lolCindy excluded, obvs.
That's fine, I'm not trying to make them like pill pockets. I'm just doing the same thing as a gelatin capsule and masking the grossness with a thin layer.
I find not many cats will consider eating pills with pill pockets on their own. Cindy excluded, obvs.
you can pick up and drop off immediately after, just do the test in your car. If you go to trowbridge you grab the kit from the attendant, pull forward 10 ft, spit, then drop it in the trash can. Or grab it at towbridge, drive to parking, spit, drop it off in front of vmc? this is all moot if your jan 4 rotation is not on campus...
Thanks. I have path next so I think I can pick it up at the VDL but then I have to go to the VMC some time before 1 to drop it off.
ah yes. they only have pick up
Fixed that for you.
There wasn’t anything to fix. I was speaking specifically in regard to the study I mentioned. I agree with you in general.
JK just got some bloodwork back that supports @LetItSnow 's hypothesisIn other news, everything keeps coming back negative so we still have no explanation for the symptoms or the eosinophilia, and I got an appointment with a specialist but not until after New Years.
Day 23 of this.
No blood at all during this whole thing, actually. It's less supportive of the UC hypothesis vs just IBD. I'm not familiar with the panel I got results for but I have a follow up appt in person tomorrow so hoping to get more info then.
Going to spoiler it in case not everyone wants to read about my fecal testing
Going to spoiler it in case not everyone wants to read about my fecal testing
Bloodwork: CBC, CMP, ESR, TTG, CRP, TSH
Ova & parasites (3x), C. diff, Giardia, fecal leukocyte staining, crypto
I switched clinics at this point b/c our campus clinic decided the answer after those labs was "we don't know" but refused to refer me, do any imaging, or do any empiric therapy, and told me to just "hang in there."
New clinic did stool testing for H. pylori, another O&P, and a multiplex PCR that was for campy, salmonella, shigella, vibrio, Y. enterocolitica, shiga toxin 1+2, norovirus, + rotavirus
Bloodwork: they used LabCorp's IBD panel, I was WNL for S. cerevisiae IgA and atypical pANCA, but S. cerevisiae IgG was 44 and looks like the ref range is 0-24.9
)
Did Puppy get into anything? Wondering about anticoagulant toxicity causing hemorrhage into the GIT (and maybe other spots).Doctoring like a boss.
My replacement came on today and said "got a case for you I took over this weekend - want to know what you'd have done".
But it was interesting to me not really because of the case itself, but because I think it highlights how 'autopilot doctoring' can sneak up on anybody, and maybe moreso in today's current world of incredibly high caseloads (my personal caseload is up about 60% this year, and it was already stressful).
For funsies, here's the case. I probably got a few details wrong, but it's basically correct.
What would you do next and why for you SA (or anyone, really) types:
13-week-old puppy presents for a 3-day history of lethargy and hyporexia and "odd behavior" that progressed to vomiting and profound hematochezia. Vaccine history is incomplete/unknown, but thought to have received at first round of a typical combo vax. Had initially seemed healthy. [I don't recall the full signalment, but it's irrelevant. If it bugs you, call it a male intact Lab.]
PE summary: Looks like a typical craptasting parvo puppy.
Init dx:
Parvo snap: "weak positive" (I'm not a fan of 'weak' anything. It's either positive or negative. But that's how it was reported, so deal with it.
CBC/Chem/Lytes highlights: HCT 24.5% (PCV 21%), Retic 500k, WBC 55k (primarily a marked neutrophilia, but I don't recall the specific diff or cytologic characteristics). Chem/Lytes WNL.
It was hospitalized as a parvo patient and tx'd with generally typical parvo care (though no NG tube was placed). Puppy improves somewhat initially, like a typical parvo patient, but its PCV continues to drop somewhat faster than is typical.
What's your next diagnostic step(s) and what's really happening?
This case is actually a pretty decent example of why all that stuff you go through in school is important. In school you think "duh, this is so easy" as you trudge through ClinPath or whatever. Out in the real (hectic, overworked) world it is super easy to get into the trap of repetition - you see a young puppy with that history, your techs ask if they can run a parvo snap, you mumble yes and move on to the next patient, you get the 'weak positive' result, and your brain shuts down when you settle on parvo as your dx. You don't think that will happen to you, because you're a super smart on-top-of-your-stuff vet student, but I guarantee you it can.
So, what's up with this puppy? Students only, no experienced clinicians allowed, especially those of you who have treated these cases.
What about newish gradsStudents only, no experienced clinicians allowed, especially those of you who have treated these cases.
I'm assuming you would have said this if it were available, but was there any imaging?Doctoring like a boss.
My replacement came on today and said "got a case for you I took over this weekend - want to know what you'd have done".
But it was interesting to me not really because of the case itself, but because I think it highlights how 'autopilot doctoring' can sneak up on anybody, and maybe moreso in today's current world of incredibly high caseloads (my personal caseload is up about 60% this year, and it was already stressful).
For funsies, here's the case. I probably got a few details wrong, but it's basically correct.
What would you do next and why for you SA (or anyone, really) types:
13-week-old puppy presents for a 3-day history of lethargy and hyporexia and "odd behavior" that progressed to vomiting and profound hematochezia. Vaccine history is incomplete/unknown, but thought to have received at first round of a typical combo vax. Had initially seemed healthy. [I don't recall the full signalment, but it's irrelevant. If it bugs you, call it a male intact Lab.]
PE summary: Looks like a typical craptasting parvo puppy.
Init dx:
Parvo snap: "weak positive" (I'm not a fan of 'weak' anything. It's either positive or negative. But that's how it was reported, so deal with it.
CBC/Chem/Lytes highlights: HCT 24.5% (PCV 21%), Retic 500k, WBC 55k (primarily a marked neutrophilia, but I don't recall the specific diff or cytologic characteristics). Chem/Lytes WNL.
It was hospitalized as a parvo patient and tx'd with generally typical parvo care (though no NG tube was placed). Puppy improves somewhat initially, like a typical parvo patient, but its PCV continues to drop somewhat faster than is typical.
What's your next diagnostic step(s) and what's really happening?
This case is actually a pretty decent example of why all that stuff you go through in school is important. In school you think "duh, this is so easy" as you trudge through ClinPath or whatever. Out in the real (hectic, overworked) world it is super easy to get into the trap of repetition - you see a young puppy with that history, your techs ask if they can run a parvo snap, you mumble yes and move on to the next patient, you get the 'weak positive' result, and your brain shuts down when you settle on parvo as your dx. You don't think that will happen to you, because you're a super smart on-top-of-your-stuff vet student, but I guarantee you it can.
So, what's up with this puppy? Students only, no experienced clinicians allowed, especially those of you who have treated these cases.
Ooooh I don't think I like where this is heading. Can I opt out and have a worm instead? Much easier to manage
What about newish grads
